penis enargement supplement

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penis enlagement pill pro solution penile enlargment testimonials vimax penis enlargement device plastic surgery penis enlarement easy enlargment free penile surgery way herbal natural penis enlargment penis enlarement pic herbal natural penis enlagement

penis enargement supplement

About 18 months ago I received an unsolicited email from a spammer trying to sell a penis enlargement product, this sounded like a dream come true but being the skeptic that I am and going by the theory of "if it sounds to good to be true it probably is" I refused to believe any sort of tablet or pump or traction device could actually make my penis a different size. After a few days though my curiosity kicked in and I decided to embark on a test to find out whether the so called enlargement products actually produce any results. I started researching the different penis enlargement products available including traction devices, pills/patches and exercise programs and recruited a team of seven friends to test them. Between us we have now tested fifteen of the best selling enlargement product/programs on the net and to our surprise yes some do actually work I must say that not all of the products we have tried worked in fact only four out of the fifteen we tried actually had any effect some of those more successful than others. The results showed that traction devices worked better than any thing else It works by the principle of traction as applied in modern medicine, for the generation of new tissue to cover burn wounds or areas of hair loss or for the lengthening of bones. I bet you are now thinking of the pain... there is none, well a little pressure when first used but no discomfort. Traction devices do have to be worn for sometime before result are seen normally between 2-4 hours a day 5 days a week. After a month you should start to see some great results. The traction system has now been medically proven to work and with continued use over a few months excellent results can be achieved. So the answer to the question is yes, do be careful though there are a lot of companies trying to sell enlargement products over the net and there are plenty of scams so keep your eyes open and do some research before you buy. free pnis enlargement technique enhancement free penis pills sample pennis enlargement video cheap penis enlargment pills top rated penis enlagement pills best penile enlargment pills vimax compare penis enlargement pills vimax penis enlargement penis enargement supplement

Erectile Dysfunction or ED (also known as Impotence) is basically seen as a man’s inability to develop or maintain an erection (of his penis) long enough for sexual relations. However it can be a warning sign of other illnesses that can be killers. Let’s look at ED How to cure it and how serious it can be to your general health. The Causes of ED There are many causes, some are psychological and some are physiological. The psychological ones may be treated in a number of ways, but the physiological ones are indicative of some other body malfunction or condition which may be serious in nature, and need medical attention. The physiological causes may be understood if there are no normal erections during sleep. A man usually has about 6 erections per sleep session, and one in the early morning when testosterone levels are at their highest. However, if these erections are not present, it can indicate some cardiovascular problem, diabetes, or hypogonadism. Hypogonadism is unacceptably low levels of testosterone in the man’s system this will affect the testicles and naturally reduce or eliminate an erection. Tests to Determine Ed If you suspect you have ED, go to a doctor, he will begin with testing your blood for diabetes, hypogonadism and prolactinoma. The doctor will also test you for cardiovascular problems, like peripheral vascular disease or coronary artery disease. He will see if you lifestyle is correct, your weight, and verify your general state of health. Simplest Test The best way to determine if your ED is psychological or physiological is to determine if you never get an erection or you do (however rare). If you get an erection at all, the causes will be found being psychological in nature. If not, you have some other causes preventing erection and this will need immediate medical attention. Current Treatment of ED Current treatment of ED can usually start with testosterone if the doctor understands there is some hormone deficiency. If there was some disease, such as diabetes, there may have been damage to the penis blood vessels. Treatment of the diabetes is essential in any event, and some of the medications below are effective for intercourse, but not a cure. ED is now treated by drugs taken orally (such as Vigara, Cialis and Leventra), injected, or even as penile suppositories. All of above will increase the efficiency of Nitrous Oxide (NO). NO dilates the blood vessels of corpora cavernosa within the penis, and allows for engorgement, and subsequent erection. Other treatments include the injections apomorphine into the erectile tissue of the penile shaft. There is now oral apomorphine as well. You can also take a number of herbs that can help increase NO and circulation to allow more blood to flow into the penis and create an erection Natural herbs to cure ED The best three are: Ginseng and Ginkgo Balboa. These have been used for thousands of years by the Chinese to improve libido and are excellent all round body tonics. And finally L Argennine – Known as natures Viagra take 2,000 mgs per day. In tests it has been proved that 80% of men who did this saw an increase in sex drive and achieve better erections. Finally, there are treatments that require surgery, such as penile implants (both permanent and inflatable). There are special vacuum pumps that can be used, that will (with the aid of an elastic band) retain a semi-erection for about 30 minutes. It is highly recommended that if you think you have ED, see your doctor at once, as it really may be the symptom of something else. Get help and get sorted your life may depend on it. pnis girth enlargement cheap penis enargement pills top rated penis enhancement pills free penile enlargment video pnis enlargement pic before and after penile enlargement forum penis enlargement review free penile enlargement technique penis enargement supplement

Have you ever felt a little concern for tigers, deers, and elephants which have been daily killed by poachers on a massive scale throughout the world for their skin, tusks and for the preparation of a variety of pharmaceutical drugs? These animals are deliberately killed for a sole purpose, to make money. Monetary greed is the only reason that the mass slaughter of animals is still proceeding unabated in spite of the authorities in various countries adopting a wealth of measures to bring such occurrence to a close. But recently it has become apparent that the trading of products obtained from animal parts has declined drastically which has brought back smiles to the faces of animal lovers across the world. A study conducted by the journal “Environmental Conservation” found that since Viagra, the erectile dysfunction cure gained entry into the pharmaceutical market after its due approval by FDA on 1998, in Hong Kong the sale of products obtained from animals to treat male impotency was decreased by 50%. Most probably you are of the extensive use of animal products for the treatment of erectile dysfunction. Hunters throughout the world have massacred animals a lot for rhinoceros horns, eggs of sea turtles and tiger penises for the utterly mistaken belief that they can sufficiently cure erectile dysfunction in men. As the Pfizer manufactured erectile dysfunction medicine Viagra started to cure erectile dysfunction patients successfully across the globe, the people who had earlier bestowed their trust on animal products to get rid of erectile dysfunction, shifted to Viagra. The manufacture of Viagra involves only chemicals and no animal product is utilized. Hence, Viagra doesn’t harm the ecological environment otherwise caused by the destruction of the principal inhabitants of the wildlife. Viagra is the first FDA (Food and Drugs Administration) approved pill for the treatment of erectile dysfunction in men and it has been proved a successful form of cure for the same from the moment it gained entry into the anti-impotency drug market. Viagra protects you from erectile dysfunction and now its role has extended in the protection of the environment as well. It is certainly a matter to celebrate even though the thought of securing the wild life only through Viagra would seem completely impractical. Yet, for the decrease in the sales of erectile dysfunction medicines produced from animal hides and other animal parts, Viagra is certainly to be credited. Viagra is an erectile dysfunction drug that works on a particular patient when he is sexually stimulated. When an erectile dysfunction patient is sexually aroused, the administration of Viagra to his system increases blood flow to his penis rapidly and as such he becomes capable of triggering off erections necessary for sufficient physical intercourse. To speak frankly, Viagra also possesses certain side-effects like any other erectile dysfunction drug which can be easily avoided. These include headache, flushing, stuffy nose, upset stomach on the occurrence of which a physician must be immediately contacted. where to buy vigrx penile enlargement secret penis enlargment excercises com enlargement penis penis pump enlargement manhattan penile surgeon enlargement free pnis pills sample extra pro solution strength natural penile enlargment technique penis enargement supplement

The terms, AIDS and HIV, have been common currency for over twenty years and in many parts of the world it can seem as if the threatened crisis never happened. We do not see the drama anticipated from a mass epidemic, but you may not be aware that more than one million adults and children are living with HIV/AIDS in the United States alone. That figure is alarming but it pales in comparison with sub-Saharan Africa, where the number affected is more than 25 million adults and children. Here are some facts on AIDS and HIV that everyone needs to understand. AIDS stands for Acquired Immunodeficiency Syndrome: • Acquired means you can get infected with it • Immune Deficiency means a weakness in the body’s fight against disease. • Syndrome means the symptoms that make up a disease. AIDS is caused by a virus called Human Immunodeficiency Virus (HIV). If you get infected with HIV, your body tries to fight the infection by producing “antibodies”. When you have a blood test for HIV, it looks for the presence and concentration of these antibodies. As the HIV disease continues, the immune system becomes weaker and, normally harmless viruses, bacteria and fungal infections can cause more sickness than if you had been healthy. You can get infected with the virus in a number of ways: • Having sex with an infected person • Sharing a needle with an infected person • Being born from an infected mother • Drinking human milk from an infected woman • Blood transfusion with infected blood The symptoms of HIV start with fever and headache, similar to influenza. The virus can meanwhile multiply in the body, damaging your immune system and making the recovery longer and longer. HIV later becomes AIDS, when the immune system is severely diminished with serious weight loss, brain tumours and other infections. There is no cure for AIDS. The drugs that are currently available simply slow the HIV damage to the Immune System. SAFER SEX HIV infection can be transmitted during sex via blood or sexual fluids from one infected person to another. Unsafe sex has a high risk of spreading HIV. When blood or sexual fluids touch the soft, moist mucous membranes inside the rectum, vagina, mouth, nose or at the tip of the penis, the HIV can enter the body. The use of condoms is vital to stop the spread of the HIV. Cuts, sores or bleeding gums increase the risk of being infected. Lubricants can increase sexual stimulation; but oil based lubricants like petroleum jelly, oils or creams can damage condoms and other latex barriers and make them ineffective as a barrier to infection. Oral sex has a high risk of transmitting HIV, especially if sexual fluids get in the mouth and there is bleeding, sores or mouth ulcers. Condoms without lubricants are best for oral sex. If you are not infected, sex with just one partner, who is also uninfected, is the safest way to stay healthy!